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It’s been a pleasure to have Chris with us throughout the series, as he’s the most knowledgeable person I know about these topics. In case you missed them, here are links to Part 1 and Part 2. The shoulder is healing up, and I’m pretty excited for our special guest today. We’ve got Chris Masterjohn back for Part 3 of the Cholesterol Series. So, in the initial parts of Part 2, what we did was looked at some of the traditional cholesterol levels in populations that have not been through industrial modernization, that have been studied and have been shown to be free of heart disease, to try to see what normal lipid metabolism is like.

In this episode, we cover: The role of cholesterol in heart disease What to do – or not do – about high cholesterol The thyroid-LDL connection and why iodine matters Are goitrogenic foods inhibiting your thyroid function and raising your cholesterol? And we looked at two groups in particular: the Masai and the Kitavans, who have been well studied and shown to be free of heart disease; and we used them to define basically the lower and upper limits of blood cholesterol.

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And this is a currently emerging field, but there is one study that was done in Japanese people, and they just looked at a number of a Japanese population that was apparently healthy, and they looked to see if they had fatty liver or not, and then they followed them over a number of years.html head meta http equiv content type text charset ISO 8859 1 title Google style body td a p h font family arial sans serif size 20px color 3366cc q 00c script function sf document f focus bgcolor ffffff 000000 link 0000cc vlink 551a8b alink ff0000 onload if images new Image src nav logo2 png topmargin 3 marginheight center div align right nowrap padding bottom 4px width 100 href url sa pref ig pval www de 3Fhl 3Dde usg Z0CJb WM4Hl Sg Uf Avcq REfrp5hx E Diese Seite personalisieren nbsp https com accounts Login continue hl Anmelden img alt height 110 intl logo gif 301 br form action search name defer table border 0 cellspacing cellpadding 4 tr b Web class imghp ie oe tab wi Bilder groups grphp wg news nwshp wn froogle frghp wf options Mehr raquo valign top 25 input hidden value maxlength 2048 55 Suche btn G submit btn I Auf gut Gl??2 advanced Erweiterte preferences Einstellungen language tools Sprachtools colspan id all radio checked label for Das lgr lr lang Seiten Deutsch cty cr country DE aus Deutschland ads Werbung services Unternehmensangebote about ? He knows more about what he’s doing right now, but he is pursuing a Ph D, and I think those of you who know his work know how much he has to bring to this discussion. Why don’t you just give a really quick intro for people who don’t already know you, and then we’ll dive in. But, what you’re saying is that what really happens is that the cholesterol — or more accurately, the lipoproteins that are carrying cholesterol and other fats — get damaged by oxidation, and then the immune system’s response to that oxidative process is what causes the buildup of plaque and then ultimately the rupture of plaque and heart attack. And what we want to do is we want to metabolize the lipids and fat-soluble nutrients and everything that’s in our bloodstream and use them properly. So, we have to keep in mind that since we’re focused on the degeneration of lipids and protecting those lipids in the blood, when we look at concentrations of lipids, we’re not trying to look at necessarily a cause-and-effect scenario. He is pursuing — well, actually I’ll let him introduce himself. So, the original theory, the infiltrative theory, is sort of like arteries are like pipes and cholesterol is like gunk, and the pipes get clogged up with cholesterol, and then you have a heart attack. That’s kind of how it was broken down in the mainstream. So, what we’re trying to do is protect the vulnerable lipids and get them to go where they need to be.

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